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Given the presence of glutathione peroxidase in meibomian glands and conjunctiva and its dependence on intracellular GSH, augmenting ocular surface redox capacity with GSH-C4 may mitigate ROS-driven epithelial stress and thereby support tear-film stability, a hypothesis-generating observation that warrants further study (Nezzar et al., 2017)

Disease-Modifying Therapies Highlight: Research Supported by Breakthrough T1D or the T1D Fund Kenneth Brayman, M.D., Ph.D., FACS (University of Virginia) presented work on two Breakthrough T1D-funded projects

Clinical data shows that most participants in tirzepatide trials regained significant weight within one year of stopping treatment

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