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We revealed that DAF-16 activation is an important factor that mediates the extended lifespan of the glp-1(-) and fem-3(-) mutants, and its forced activation can reverse the shortened lifespan of the mog-3(-) mutant

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Neuroprotection : Enhancing mitochondrial function and reducing free-radical production under hypoxic conditions ( 9, 10 )

BPC-157 operates locally as an **ultra-stable cytoprotectant**: it survives gastric milieu, reactivates the VEGF-receptor-2 pathway at the lesion site, modulates the nitric oxide system (NO / eNOS), upregulates growth hormone receptors on tendon fibroblasts (Chang et al