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2.2 Neurotoxic mechanisms METH induces neurotoxicity via a combination of oxidative stress, mitochondrial dysfunction, excitotoxicity, and neuroinflammation
Further studies have shown that the myristoylation of FSP1 mediates the recruitment of this protein to the plasma membrane, where it performs its function as an oxidoreductase that reduces COQ10 (also known as ubiquinone-10) and as a lipophilic radical-trapping antioxidant that halts the propagation of lipid peroxides
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2025 Exhibit 131: Percentage of Covered Workers with Pharmacy Benefit Deductibles, Employer-Sponsored Plans, 2005 to 2025 Exhibit 132: Enrollment in Individual Insurance Market, by Source of Plan, 2015 to 2025 Exhibit 133: Type of Cost Sharing for Prescription Drug Benefits, Silver Health Insurance Marketplace Plans, 2025 Exhibit 134: Average Cost Sharing by Prescription Drug Tier, Silver Health Insurance Marketplace Plans, 2025 Exhibit 135: Presence of Separate Prescription Drug Deductibles in Silver Plans on Health Insurance Marketplaces, 2015 to 2025 Exhibit 136: Medicare Part D Enrollment, by Type of Plan, 2010 to 2025 Exhibit 137: Standard Medicare Prescription Drug Benefit, 2025 Exhibit 138: Number of Stand-Alone Medicare Part D Prescription Drug Plans, 2007 to 2026 Exhibit 139: Monthly Rebate per Medicare Advantage Beneficiary, by Extra Benefit Type, 2020 to 2026 Exhibit 140: Type of Cost Sharing for Prescription Drug Benefits, Medicare Part D Plans, 2025 Exhibit 141: Median Cost Sharing by Prescription Drug Tier, Medicare Part D PDPs, 2025 Exhibit 142: Median Cost Sharing by Prescription Drug Tier, Medicare Advantage Plans, 2025 Exhibit 143: Median Cost Sharing Amounts for 12 Largest Medicare Part D Plans, 2025 Exhibit 144: Summary of Plan Sponsors Tools for Accessing Manufacturer Patient Support Funds to Offset Drug Spending Exhibit 145: Prevalence of Copayment Offset Programs for Specialty Drugs, 2020 vs
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